News
In this section you will find Society press releases, member announcements, and ISN communication products.
ISN Announcements
Renew your ISN Membership at ASN Renal Week 2009
21 October 2009
By renewing your ISN membership you reiterate your decision as a concerned and engaged citizen to do something for your fellow nephrologists with fewer resources.
Renew while at the ASN Renal Week and receive a free ISN Travel Mug! ISN will be welcoming you at the ISN booth 414-416 on 29-31st October.
Check out the new World Kidney Day website!
19 October 2009
Visit and populate the new World Kidney Day website to make sure that next WKD is a huge success!
This is now YOUR website!
ISN Press Room
Pakistan: Cutting Down on 'Kidney Tourism'
22 February 2008
Pakistan’s Transplantation of Human Organs and Human Tissue Ordinance 2007 mandates punitive measures for those who deal in organs or remove them improperly. More importantly, the ordinance explicitly bans the donation of organs to foreigners. Opportunities to donate organs to non-relatives are also curtailed.
Renal News
Small Increases In Phosphorus Mean Higher Risk Of Heart Disease
Urology / Nephrology News From Medical News Today : November 07, 2009
Higher levels of phosphorus in the blood are linked to increased calcification of the coronary arteries a key marker of heart disease risk, according to a study in an upcoming issue of Clinical Journal of the American Society of Nephrology (CJASN). "This may help to explain why even early-stage chronic kidney disease (CKD) is associated with increased cardiovascular risk that is not otherwise explained by traditional risk factors," comments Katherine R.
Polycystins: Proteins That Regulate The Cellular Barometer
Urology / Nephrology News From Medical News Today : November 07, 2009
What is the role of proteins called polycystins in patients with polycystic kidney disease? A team of researchers from CNRS and INSERM, led by Eric Honore from the Institut de Pharmacologie Moléculaire et Cellulaire (IPMC, Universite de Nice Sophia Antipolis/CNRS) has elucidated the molecular and cellular mechanisms linked to polycystin malfunctions that cause this common hereditary disease.
